REDUCING FALSE RECOGNITION IN THE DEESE-ROEDIGER/MCDERMOTT PARADIGM: RELATED LURES REVEAL HOW DISTINCTIVE ENCODING IMPROVES ENCODING AND MONITORING PROCESSES

Reducing False Recognition in the Deese-Roediger/McDermott Paradigm: Related Lures Reveal How Distinctive Encoding Improves Encoding and Monitoring Processes

In Toys the Deese-Roediger/McDermott (DRM) paradigm, distinctive encoding of list items typically reduces false recognition of critical lures relative to a read-only control.This reduction can be due to enhanced item-specific processing, reduced relational processing, and/or increased test-based monitoring.However, it is unclear whether distinctive

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A Rare Cause of Neonatal Hypotonia: First Case of Autosomal Recessive Fast-Channel Congenital Myasthenic Syndrome Type 1B in Albania

Hypotonia is a concern, with high morbidity and poor outcomes in 10% of neonatal care cases.Neonatal hypotonia presents a diagnostic challenge for neonatologists, as it may be a sign of a central nervous disorder (hypoxic-ischemic Gift Cards insult, intracranial hemorrhage, cerebral palsy), inborn errors of metabolism, a primary neuromuscular disor

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High-Rise Construction in Densely Dwelled Cities: Requirements for Premises Insolation and Consequences of their Violation in Russian Law and Jurisprudence

The issues of constructing high-rise, primarily residential, buildings have a great social significance.Not every plot of land, acquired in the Russian Federation is suitable for high-rise construction.Therefore, every construction company that plans to erect a multi-apartment building, a high-rise office building, or a skyscraper must take into ac

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Canine disorder mirrors human disease: exonic deletion in HES7 causes autosomal recessive spondylocostal dysostosis in miniature Schnauzer dogs.

Spondylocostal dysostosis is a congenital disorder of the axial skeleton documented in human families from diverse racial backgrounds.The Honey condition is characterised by truncal shortening, extensive hemivertebrae and rib anomalies including malalignment, fusion and reduction in number.Mutations in the Notch signalling pathway genes DLL3, MESP2

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